Findings:
Compromised image quality due to motion artifacts.
No intra- or extra-axial mass lesion or collection is identified. The ventricles and sulcal spaces are within normal limits. There is no midline shift.
Confluent zones of T2/Flair hyperintensity involving the subcortical and periventricular cerebral white matter, also involving the basal ganglia on the brain stem, greater than expected for age, in keeping with advanced small vessel ischaemic disease.
Multiple, chronic lacunar infarctions are seen in bilateral centrum semiovale, the basal ganglia, both thalami and the pons.
Small postischaemic defects are seen in the PICA territory in both cerebellar hemispheres. No diffusion restriction.
Numerable foci of blooming artefact in keeping with blood products, are present in both cerebral hemispheres as well as in the left thalamus.
The orbits are clear. Partial obliteration of the mastoid air cells on the right side is seen. Mild mucosal thickening is seen in the paranasal sinuses.
No bony abnormality is seen.
Conclusion:
No evidence of an acute/subacute ischaemic event.
Extensive small vessel ischaemic disease as well as multiple lobar/cortical microbleeds in both cerebral hemispheres, in keeping with cerebral amyloid angiopathy.